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Caspase-1 promotes endothelial progenitor cells(EPC) undergo pyroptosis and weakens EPC’s angiogenesis after myocardiac infarction

Ren GongXiao HuangYing YinXiaohua JiangXinyuan Lierhe Gaohongyu ZhangSteven HouserNicholas E.HoffmanMuniswamy MadeshCraig ThomasHong WangXiao-feng Yang

Cardiovascular Research Center,Center for Translational Medicine,Temple University School of MedicineDepartment of Pharmacology,Temple University School of MedicineThrombosis Research Center,Temple University School of MedicineDepartment of Biochemistry,Temple University School of MedicineDepartment of Cardiology,Jiangxi People’s HospitalDepartment of Cardiology,The second affiliated hospital to Nanchang UniversityNIH Chemical Genomics Center,National Human Genome Research Institute,National Institutes of Health8,Bethesda,MD 20892-3370Department of Pharmacology2,Temple University School of MedicineThrombosis Research Center3,Temple University School of MedicineDepartment of Biochemistry7,Temple University School of MedicineNIH Chemical Genomics Center,National Human Genome Research Institute,National Institutes of Health8,Bethesda,MD 20892-3370,USA

摘要:<正>We examined an important question whether endothelial progenitor cells(EPCs) have capase-1 pathway,which can sense dyslipidemia,a risk factor for cardiovascular disease,as a metabolic danger-associated molecular pattern(DAMP),and activat EPC death and weaken EPC repairing.Using techniques including immunological,biochemical,gene deficient mice,
会议名称:

第15届中国南方国际心血管病学术会议

会议时间:

2013-04-11

会议地点:

中国广东广州

  • 专辑:

    医药卫生科技

  • 专题:

    心血管系统疾病

  • 分类号:

    R542.22

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